Diabetes Mellitus and Food Balance in the Kingdom of Saudi Arabia

The present explanatory study concerns with the relation between Diabetes Mellitus and Food Balance in the Kingdom of Saudi Arabia during 2005-2010, using published data. Results illustrated that Saudi citizen daily protein consumption (DPC) during 2005-2007 (g/capita/day) is higher than the average global consumption level of protein with 15.27%, daily fat consumption (DFC) with 24.56% and daily energy consumption (DEC) with 16.93% and increases than recommended level by International Nutrition Organizations (INO) with 56% for protein, 60.49% for fat and 27.37% for energy. On the other hand, DPC per capita in Saudi Arabia decreased during the period 2008-2010 from 88.3 to 82.36 gram/ day. Moreover, DFC per capita in Saudi Arabia decreased during the period 2008-2010 from 3247.90 to 3176.43 Cal/capita/ day, and daily energy consumption (DEC) of Saudi citizen increases than world consumption with 16.93%, whereas increases with 27.37% than INO. Despite this, DPC, DFC and DEC per capita in Saudi Arabia still higher than world mean. On the other side, results illustrated that the number of diabetic patients in Saudi Arabia during the same period (2005-2010). The curve of diabetic patient’s number in Saudi Arabia during 2005-2010 is regular ascending with increasing level ranged between 7.10% in 2005 and 12.44% in 2010. It is essential to devise Saudi National programs to educate the public about the relation of food balances and diabetes so it could be avoided, and provide citizens with healthy dietary balances tables.

Authors:

• Aljabryn Dalal Hamad

Keywords:

• Diabetes Mellitus
• Food Balance
• Energy
• Fat
• Protein
• Saudi Arabia.

References:

[1] IDF–International Diabetes Federation. Facts and Figures:
http://www.idf.org/home/index.cfm?node=37.
[2] Marble, A.; White, P.; Bradley, R.F. and Krall, L.P.(eds). (1973).
Joslin’s Diabetes mellitus, Philadelphia: Lea and Fabiger, 1-5.
[3] Irvine, W .J. (1977).Classification of idiopathic diabetes. Lancet, i: 638-
642.
[4] National Diabetes Data Group (1979). Classification and diagnosis of
diabetes mellitus and other categories of glucose intolerance. Diabetes,
28: 1039-1057.
[5] El-Hazmi, M.; Warsy, A.; Al-Swailem, A.; Al-Swailem, A. and
Sulaimani, R. (1998).Diabetes mellitus as a health problem in Saudi
Arabia. East Mediterr. Health J., 4: 58-67.
[6] Diabetes Atlas (2012). Global and Regional Diabetes Statistics.
[7] Bonadonna, R.C., Groop, L. and Kraemer, N.(1990). Obesity and insulin
resistancein humans: a dose-response study. Metabolism. 39:452–459.
[8] Rexrode, K. M., Manson, J. E. and Hennekens, C. H. (1996). Obesity
and cardiovascular disease. Curr. Opin. Cardiol. 11:490–495.
[9] FAO/WHO/UN (1985). Energy and protein requirements. Report
FAO/WHO/UN Expert Consultation. Geneva, World Health
Organization, Technical report Series, No. 724.
[10] Beaton, G. H. (1991). Human nutrient requirement estimates. Food
nutrition and Agriculture, Vol.1: 3-15.
[11] Khan, M. A. (1991). Recommended dietary allowances for Pakistan,
Islamabad. Pakistan National Agricultural Research Center.
[12] Scrimshaw, N.S. (1996). Human protein requirements: a brief update,
Food and nutrition bulletin, 17 (3): 185-190.
[13] NCEP (2002). Third Report of the National Cholesterol Education
Program Expert Panel on Detection, Evaluation, and Treatment of High
Blood Cholesterol in Adults (Adult Treatment Panel III): final report.
Circulation 106:3143–3421.
[14] Mensah, G. A.; Mokdad, A. H.; Ford, E; Narayan, K. M.; Giles, W. H.;
Vinicor, F. and Deedwania, P. C.(2004). Obesity, metabolic syndrome,
and type 2 diabetes: emerging epidemics and their cardiovascular
implications. CardiolClin 22:485–504.
[15] Chan, J. M.; Rimm, E. B.; Colditz, G. A.; Stampfer, M. J. and Willett,
W. C. (1994). Obesity, fat distribution, and weight gain as risk factors
for clinical diabetes in men. Diabetes Care 17:961–969.
[16] Rexrode, K. M.; Carey, V. J.; Hennekens, C. H.; Walters, E. E.; Colditz,
G. A.; Stampfer, M. J.; Willett, W. C. and Manson, J. E. (1998).
Abdominal adiposity and coronary heartdisease in women. JAMA
280:1843–1848.
[17] Badran, M. and Laher, I. (2011). “Obesity in 3iarrh-speaking
countries,”Journal of Obesity, vol. 2011, Article ID 686430, 9 pages.
[18] National Center for Health Statistics (1987).Druny, T.F. and Powell,
A.L.: Prevalence of known diabetics among Black Americans. Advance
data from vital and health statistics. Hyattsville MD, Public Health
Service. DHHS Publication No. 130 (PHS) 87-1250.
[19] McKinley Health Center (2008). Macronutrients: The Importance of
Carbohydrate, Protein, and Fat.
[20] Ulmer, G. (2010). Diet and Nutrition. Recommended daily Nutritional
intake for health. Livestrong. [21] Spenser, K. and Cudworth, A. (1983).The aetiology of insulin dependent
diabetes mellitus. In: Mann JI,Pyorala K, Teuscher A (Eds). Diabetes in
Epidemiological Perspective. Churchill Livingstone, London.
[22] Hinsworth, H. P. (1935). Dietatic factor determining glucose tolerance
and sensitivity to insulin of healthymen.ClinSci, 2: 67-94.
[23] Yudkin, J. (1964). Dietary fat and dietary sugar in relation to ischaemic
heart disease and diabetes. Lancet, 2: 4-5.
[24] Tsai, S. H. (1971). Sugar consumption in Taiwan. In: Tsuji S, Wadar M.
Diabetes Mellitus in Asia. Amsterdam, Excerpta Med: pp 104.
[25] Trowell, H. P. (1974). Diabetes mellitus death – rates in England and
Wales 1920-70 and food supplies. Lancet, 2: 998-1004.
[26] Winterbotham, H. J. (1961).Diabetes mellitus at Mabuiag Island, Torres
Straits, 1960. Med J Aust., I:780-781.
[27] West, K. M. (1978). Epidemiology of diabetes and its vascular lesion.
New York, Elsevier, pp: 19.
[28] Short, J. J. and Johnson, H. J. (1939). Glucose tolerance in relation to
weight and age: Study of 541 cases. ProcAssoc Life Ins Med Dire, 25:
237-257.
[29] Vinke, B.; Nagelsmit, W. F. and Buchan-Van F. S. (1959).Some
statistical investigations in diabetes mellitus. Diabetes, 8: 100-104.
[30] Slome, C.; Gampel, B.; Abramson, J. H. and Scotch, N. (1960). Weight,
height and skinfold thickness of Zulu adultsin Durban. S. Afr Med J, 34:
505-509.
[31] Jackson, W. P. (1978). Epidemiology of diabetes in South Africa.
Advance Metabol. Discord, 9: 111-146.
[32] Mann, J. I.; Pyorala, K. and Teuscher, A. (1983). Diabetes in
epidemiological perspective. Churchill Livingstone, London.
[33] Ohlson, L. O.; Larsson, B. and S’Vardsudd, K.(1985).The influence of
body fat distribution on the incidence of diabetes mellitus. 13:5 years of
follow-up of the participants in the study of members in 1913. Diabetes,
34: 1055-1058.
[34] Joslin, E. P. (1972). The prevention of diabetes mellitus. J Amer Med
Assoc, 76: 79-84.
[35] Kingston, M. and Skoog, W. C. (1986). Diabetes in Saudi Arabia. Saudi
Med J,7(2): 130-142.
[36] Taylor, R.; Ram, P. and Zimmet, P. (1984).Physical activity and
prevalence of diabetes in Melanesian and Indians males in Fiji.
Diabetologia, 27: 578-582.
[37] Ministry of Agriculture (2012). Food Balance Sheets for the period
(2005-2007) to (2008-2010). Agricultural Research and Development
Affairs, Dept. of Studies Planning and Statistics, 7th Issue: pp 78.
[38] Al Hameed, M. S. (2007). Diabetes Mellitus, Causes, complications and
treatment. King Saud University, Faculty of Medicine. Kingdom of
Saudi Arabia, Riyadh.